Genetics Society of America
GSA connects you to the field through a range of scholarly and community publications: our peer-reviewed journals offer two distinct platforms for communicating high-quality original research; our blog brings perspective to your research and career; and our newsletters keep you up to date with the latest opportunities and news from your community.
GENETICS publishes high-quality genetics and genomics research that expands scientific boundaries—we’ve been building the field since 1916. With its broad readership, rich history, and responsive editors, GENETICS brings the latest in publishing innovations to the communities it serves. We invite you to submit your research and discover the fast turnaround times and helpful review process for yourself.
Get your useful data out into the world by publishing in G3: Genes|Genomes|Genetics. G3 publishes high quality foundational research, particularly studies that generate useful genetic information, such as mutant screens, single gene studies, genome maps, genome sequence data, GWAS and QTL studies, software, data resources, and new methods. The Editorial Board of G3 believes that rapid dissemination of such data lays the foundation for many important insights.
We treat authors as colleagues, not adversaries. Your time is important—and so is your research. Peer editors oversee the review process and give you clear guidance on how to address any reviewer comments; our helpful editorial staff keep the process moving and on time. We make it easy to submit: no formatting requirements on initial submission. Our dedicated editors work hard to keep time to first decision at around a month. And because we’re agile and community-driven, we’ve long been at the forefront of publishing innovations.
The GSA Journals are committed to helping authors get their work in front a wide audience. Our Promotional Toolkit for Authors contains pointers to help you promote your GENETICS or G3: Genes|Genomes|Genetics paper, including how GSA and Oxford University Press can help.
The review experience has been the single most useful and pleasant of my career. The reviews were constructive and fair, the editor provided exceptionally clear guidance, and turn-around on the revision was lightning fast.
Genes to Genomes, the GSA blog, features news from the GSA community, highlights from the GSA journals, and posts from staff and guest authors about careers, professional development, science policy, publishing, education, outreach, equity and inclusion, and the intersections of science, art, and culture. Propose a guest post yourself and start a conversation!
In March 2016, GENETICS launched WormBook, a comprehensive compendium of review articles presenting the current state of knowledge in C. elegans research. WormBook articles span the breadth of the biology, genetics, genomics, and evolutionary biology of C. elegans. Find historical WormBook articles published as the online companion to WormBase here. This page contains the full list of all historical and GSA-published WormBook articles, organized by section.
G3 is expanding its report formats to make it faster and easier for researchers to share high-value datasets, tools, methods, and screening results with the scientific community. From Genetic Screen Reports to new Molecular Method...
AbstractMachine learning (ML) models with stochastic and non-deterministic characteristics are increasingly used for genomic prediction in plant breeding, but evaluation often neglects important aspects like prediction stability and ranking performance. This study addresses this gap by evaluating how two hyperparameters of a Gradient Boosting Machine (GBM), learning rate (v) and boosting rounds (ntrees), impact stability and multi-metric predictive performance for cross-season, cross-environment prediction in a MAGIC wheat population. Using a grid search of 36 parameter...
AbstractCandida albicans is an opportunistic fungal pathogen and a component of the human microbiome. C. albicans virulence traits include biofilm production, which is governed by a large transcriptional network. Mutations of some biofilm regulators cause the same severe biofilm-defective phenotype in multiple clinical isolates. Mutations of others, such as Wor3, Bcr1, Ndt80, and Ume6, have mild or variable phenotypes among clinical isolates. We hypothesized that Wor3 may share functions with another variable-phenotype biofilm regulator. This...
